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Jonathan M. Green, MD

Professor, Medicine
Division of Pulmonary and Critical Care Medicine
Professor, Pathology and Immunology

Specialty Areas

Critical Care Medicine
Pulmonary Disease
Internal Medicine

Board Certifications

Critical Care Medicine
Internal Medicine
Pulmonary Disease

Hospital Affiliations

Barnes-Jewish Hospital

Areas of Clinical Interest

Internal medicine, critical care medicine, pulmonary medicine, airway inflammation, inflammatory lung disease

  • Education
  • Publication & Research

Education

Education

Fellowship : Pulmonary, University of Michigan, Ann Arbor, Michigan 1993
Residency: Boston City Hospital, Boston Massachusetts 1989
Medical Degree: Wayne State University School of Medicine, Detroit Michigan 1986
B.S.: University of Michigan, Ann Arbor, Michigan 1982

Publication & Research

Publication & Research

CD28 Promotes Plasma Cell Survival, Sustained Antibody Responses, and BLIMP-1 Upregulation through Its Distal PYAP Proline Motif. Rozanski CH, Utley A, Carlson LM, Farren MR, Murray M, et al.Journal of immunology (Baltimore, Md. : 1950). 2015; 194(10):4717-28.

Interleukin-7 and anti-programmed cell death 1 antibody have differing effects to reverse sepsis-induced immunosuppression. Shindo Y, Unsinger J, Burnham CA, Green JM, Hotchkiss RS. Shock (Augusta, Ga.). 2015; 43(4):334-43.

Costimulation via the tumor-necrosis factor receptor superfamily couples TCR signal strength to the thymic differentiation of regulatory T cells. Mahmud SA, Manlove LS, Schmitz HM, Xing Y, Wang Y, et al. Nature immunology. 2014; 15(5):473-81.

Cutting edge: A double-mutant knockin of the CD28 YMNM and PYAP motifs reveals a critical role for the YMNM motif in regulation of T cell proliferation and Bcl-xL expression. Boomer JS, Deppong CM, Shah DD, Bricker TL, Green JM. Journal of immunology (Baltimore, Md. : 1950). 2014; 192(8):3465-9.

Reactivation of multiple viruses in patients with sepsis. Walton AH, Muenzer JT, Rasche D, Boomer JS, Sato B, et al. PloS one. 2014; 9(2):e98819.

The changing immune system in sepsis: is individualized immuno-modulatory therapy the answer? Boomer JS, Green JM, Hotchkiss RS. Virulence. 2014; 5(1):45-56.

A detailed phenotypic analysis of immune cell populations in the bronchoalveolar lavage fluid of atopic asthmatics after segmental allergen challenge. Boomer JS, Parulekar AD, Patterson BM, Yin-Declue H, Deppong CM, et al. Allergy, asthma, and clinical immunology : official journal of the Canadian Society of Allergy and Clinical Immunology. 2013; 9(1):37.

CTLA4Ig inhibits effector T cells through regulatory T cells and TGF-β. Deppong CM, Bricker TL, Rannals BD, Van Rooijen N, Hsieh CS, et al. Journal of immunology (Baltimore, Md. : 1950). 2013; 191(6):3082-9.

CD43-mediated IFN-γ production by CD8+ T cells promotes abdominal aortic aneurysm in mice. Zhou HF, Yan H, Cannon JL, Springer LE, Green JM, et al. Journal of immunology (Baltimore, Md. : 1950). 2013; 190(10):5078-85.

Blockade of the negative co-stimulatory molecules PD-1 and CTLA-4 improves survival in primary and secondary fungal sepsis. Chang KC, Burnham CA, Compton SM, Rasche DP, Mazuski RJ, et al. Critical care (London, England). 2013; 17(3):R85.